198c9b8daecc44fbda6a6494c566c723920f030a
lrnassar
Wed Mar 11 18:25:21 2026 -0700
Fixing a few hundred clear typos with the help of Claude. Some are less important in code comments, but majority of them are in user-facing places. I manually approved 60%+ of the changes and didn't see any that were an incorrect suggestion, at worst it was potentially uncessesary, like a code comment having cant instead of can't. No RM.
diff --git src/hg/makeDb/trackDb/human/hg17/cnp.html src/hg/makeDb/trackDb/human/hg17/cnp.html
index e26997edb24..d0140b6b60b 100644
--- src/hg/makeDb/trackDb/human/hg17/cnp.html
+++ src/hg/makeDb/trackDb/human/hg17/cnp.html
@@ -254,31 +254,31 @@
genetic polymorphism.
Conrad genotype analysis
The authors first tested 12 predicted deletions using quantitative PCR.
For all 12 deletions, DNA concentrations consistent with transmission of a
deletion from parent to child were observed.
To provide more extensive validation by comparative genome hybridization (CGH), the authors designed a
custom oligonucleotide microarray comprised of 380,000 probes that tile across all 134 candidate deletions
identified in 9 HapMap offspring (8 YRI and 1 CEU).
The results of this CGH analysis indicate that the majority (about 85%) of candidate deletions detected
by the method are real.
Redon analysis of HapMap data
-The authors utilized numerous quality meaures, including
+The authors utilized numerous quality measures, including
repeated experiments on the WGTP array for 82 individual and on the 500K EA
array for 15 individuals.
The average false-positive rate per experiment was held beneath 5%. Aberrant chromosomes were
removed from the analysis. Further details are available in the Nature paper cited below.
References
Conrad, D., Andrews, T.D., Carter, N.P., Hurles, M.E., Pritchard, J.K.
A high-resolution survey of deletion polymorphism in the human genome.
Nature Genet 38(1), 75-81 (2006).
Hinds, D., Kloek, A.P., Jen, M., Chen, X., Frazer, K.A.
Common deletions and SNPs are in linkage disequilibrium in the human genome.
Nature Genet 38(1), 82-85 (2006).