198c9b8daecc44fbda6a6494c566c723920f030a lrnassar Wed Mar 11 18:25:21 2026 -0700 Fixing a few hundred clear typos with the help of Claude. Some are less important in code comments, but majority of them are in user-facing places. I manually approved 60%+ of the changes and didn't see any that were an incorrect suggestion, at worst it was potentially uncessesary, like a code comment having cant instead of can't. No RM. diff --git src/hg/makeDb/trackDb/human/hg18/cnp.html src/hg/makeDb/trackDb/human/hg18/cnp.html index b75f4614a38..1f6a2657ac1 100644 --- src/hg/makeDb/trackDb/human/hg18/cnp.html +++ src/hg/makeDb/trackDb/human/hg18/cnp.html @@ -232,31 +232,31 @@ matched prediction across a set of 12-24 individuals. The authors confirmed 51 of 60 candidate variants by this criterion.
The authors performed quantitative PCR in all 269 HapMap DNA samples for 11 candidate deletions that overlapped the coding exons of genes and that were discovered in many individuals. At 10/11 loci, the authors observed three discrete clusters, identifying individuals with zero, one and two gene copies. All 60 trios displayed Mendelian inheritance for the ten deletions, as well as Hardy-Weinberg equilibrium in all four populations surveyed, and transmission rates close to 50%. This suggests that the deletions behave as a stable, heritable genetic polymorphism.
Thanks to Lars Feuk at The Hospital for Sick Children in Toronto for providing these data in hg18 coordinates.