4ea3d549b2d0aa682aa22e6b31e844fee78d2cfc
gperez2
  Tue Oct 21 13:55:33 2025 -0700
Code review edit, refs #36551

diff --git src/hg/makeDb/trackDb/human/spliceImpactSuper.html src/hg/makeDb/trackDb/human/spliceImpactSuper.html
index c2f3d795d29..0b0748f94bc 100644
--- src/hg/makeDb/trackDb/human/spliceImpactSuper.html
+++ src/hg/makeDb/trackDb/human/spliceImpactSuper.html
@@ -101,31 +101,31 @@
 </ul>
 </p>
 Mouseover on items shows the variant, gene name, type of change (donor gain/loss, acceptor
 gain/loss), location of affected cryptic splice, and spliceAI score. Clicking on any item brings up
 a table with this information.
 </p>
 <p>
 The scores range from 0 to 1 and can be interpreted as the
 probability of the variant being splice-altering. In the paper, a detailed characterization is
 provided for 0.2 (high recall), 0.5 (recommended), and 0.8 (high precision) cutoffs.</p>
 
 <h3>SpliceAI Wildtype</h3>
 <p>
 These tracks are in bigWig format. The signal height represents the SpliceAI probability score.
 This track may be configured in a variety of ways to highlight different aspects of the displayed
-information. Click the "Graph configuration help" link for an explanation of configuration
+information. Click the &quot;Graph configuration help&quot; link for an explanation of configuration
 options.</p>
 
 <h3>SpliceVarDB</h3>
 <p>According to the strength of their supporting
 evidence, variants were classified as &quot;splice-altering&quot; (~25%), &quot;not
 splice-altering&quot; (~25%), and &quot;low-frequency splice-altering&quot; (~50%), which
 correspond to weak or indeterminate evidence of spliceogenicity. 55% of the
 splice-altering variants in SpliceVarDB are outside the canonical splice sites
 (5.6% are deep intronic). The data is shown as lollipop plots that can be clicked, 
 the details page then shows a link to SpliceVarDB with full details.
 </p>
 
 <p>The classification thresholds primarily follow those established by the original study.
 However, most studies only defined criteria for splice-altering variants and did not define
 criteria for variants that resulted in normal splicing. The authors implemented stringent